"detect that autism is caused by a miscommunication neural
Source: detect that autism is caused by a miscommunication neuronal
Monday, March 28, 2011
Mario Salieri En Español On Line
An experiment in mice at the Massachusetts Institute of Technology (MIT) and published in Nature this week, confirms the theory that autism and all related diseases could be linked to the defect of a protein whose mission is to link neurons together. At work , In which participated the primary computer, the MIT, one of Duke University (North Carolina) and one from the University of Coimbra, Portugal, studied the behavior of mice to which they had been tampered with SHANK3 gene, and found that acting in a compulsive and self-harm in relation to cleaning and avoided contact with others, as with people suffering from this disease. A gene known as The affected gene was investigated in the past in relation to autism. In 2007, fieldwork of the Pasteur Institute looked at a group of families with sick and found an association between mutations and congenital SHANK3. However, failed to establish a cause-effect remained a mystery biological principle of this group of diseases, suffering varying degrees six of every thousand children. The key may have given the doctor Guoping Feng, the director's most recent work. For the MIT researcher could be several genes that are related to the functioning of the synapse-related protein-intercom-neurons. The mice studied were found defects in the circuits connecting different areas of the brain, cortical and striatal regions. Feng understand why the connections between these areas could be the key to regulating the behavior and interaction social. Many proteins, a failure Guoping Feng said about the BBC: "These findings allow us to know what defects the neural circuitry required for these abnormal behaviors, which could lead to new strategies for the development of a treatment." This would, predicts Feng, the ability to restore synaptic function, neuronal intercom, "regardless of the specific protein that is damaged. The task, simple on paper, does not seem feasible in the short term due to the uniqueness of the brain, but it marks the way forward for future research.
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